Page 6 of articles about pcv2

It is believed that TTSuVs themselves do not directly cause any disease but they could potentiate coinfecting pathogens and participate as triggering factors for diseases like PCVDs and PRDC.

PRRSV by itself may induce mild-to-severe respiratory disease but rarely results in mortality.

PCV2 may reach embryos/foetuses upon transplacental spread during viremia or upon insemination with contaminated semen.

It has been shown that the efficacy of PCV2 vaccines was dramatically decreased in the context of husbandry deviations such as excess of cross-fostering, mingling at weaning or very large groups in nursery.

One month after the beginning of the outbreak of reproductive failure in sows, acute respiratory disorder and diarrhoea that was non-responsive to antibiotics started in weaned pigs aged 6-8 weeks...

The efficacy of PCV2 vaccines to control PCV2 associated diseases has been clearly demonstrated not only in piglets but also in sows. Moreover, it is also feasible to vaccinate sows and their offspring without hampering its growth during the rearing period.

The prevalence of pleurisy is surprisingly high among pigs at slaughter. A recent review of available data ranged from 12.5% in the UK, 26% in Spain, to 41% of individual pigs slaughtered in one Norwegian study.

Experimental Mhyo and PCV2 co-inoculations have resulted in somewhat divergent results.

PCV2 can be shed into semen by infected boars and research suggests that the virus can then be transmitted to gilts and sows by AI causing reproductive failure but there is no evidence that PCV2 in semen has effects on various characteristics of sperm cells.